Polyphenol / Sirtuin Activator

Resveratrol

A polyphenolic stilbenoid found in red wine and grape skins that activates SIRT1 and AMPK pathways. Requires NAD+ co-factors for maximal sirtuin activation, making it synergistic with NMN.

longevitycardiovascularanti-inflammatory
Tier AWell-tolerated — strong human evidence
Evidence gradeBControlled trials / Cohort studies
JS

Reviewed & fact-checked by

Dr. Jane Smith, MD, PhD

Chief Medical Reviewer · Last updated: January 15, 2026

Verified

Mechanism of Action

Resveratrol (3,5,4′-trihydroxystilbene) is a polyphenolic compound produced by plants under stress conditions. Its primary longevity-related mechanisms include:

  • SIRT1 activation: Resveratrol acts as a SIRT1-activating compound (STAC), allosterically activating SIRT1 deacetylase activity. SIRT1 requires NAD+ as a co-substrate, which is why co-supplementation with NMN produces synergistic effects. []
  • AMPK activation: Resveratrol activates AMPK (AMP-activated protein kinase), the master metabolic sensor, mimicking some aspects of caloric restriction
  • Anti-inflammatory: Inhibits NF-κB signalling, reducing inflammatory cytokine production
  • Antioxidant: Scavenges reactive oxygen species via its polyphenol hydroxyl groups

Bioavailability Challenge

Standard resveratrol has notoriously poor oral bioavailability (~1%) due to rapid hepatic conjugation. Key strategies to improve absorption:

  • Take with fat: Co-ingestion with a fat-containing meal increases bioavailability 5-fold
  • Micronised formulations: Particle reduction increases surface area and absorption rate
  • Liposomal delivery: Enhanced bioavailability in preliminary studies
  • Trans- vs cis-isomer: trans-resveratrol is the biologically active form — verify this in product COAs

Clinical Evidence Summary

A 2012 meta-analysis of 11 RCTs found significant improvements in fasting glucose, insulin, and blood pressure in type 2 diabetic patients. [] A 2020 systematic review noted benefits in cardiovascular biomarkers, though acknowledged the bioavailability challenge as a limiting factor across studies. []

Stacking Interactions

How Resveratrol interacts with other compounds

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NMNSynergisticmoderate evidence

Classic Sinclair protocol: take together in the morning with a fat-containing meal. Fat improves resveratrol bioavailability significantly.

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QuercetinSynergisticweak evidence

Both are poorly bioavailable without fat. Take together at meals.

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TMG (Trimethylglycine)Neutralanecdotal evidence

No significant direct interaction. Can be taken together without concern.

Safety Profile — Tier A

Well-tolerated — strong human evidence

Contraindications

  • Blood-thinning medications (warfarin, aspirin) — may potentiate anticoagulant effect
  • Hormone-sensitive cancers (oestrogen-receptor positive)
  • Pregnancy — insufficient safety data

Side Effects

  • Gastrointestinal discomfort at high doses (>1g/day)
  • Mild headache in some users
  • Nausea if taken without fat source (poor absorption)

Drug Interactions

Warfarin / Coumadin (increased bleeding risk)Cyclosporine (CYP3A4 inhibition)Statins (may increase statin blood levels)